By David B. Rubin M.D. (auth.), Peter Polgar (eds.)
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Additional resources for Eicosanoids and Radiation
From basics it is, therefore. easy to understand. that the presence of elevated blood lipids 8I1d in particular of cholesterol would favour the foam cell formatiOJl (43) especially insofar as deaeased local POll-levels are favouring inIracellular cholesterol-synthesis and decrease the intracellular degndatiOJl (23). Furthermore. malondialdehyde (ftpe 8) derived from activated pllleletstogether withLDLresultinginmalonyl-LDL(I7)formationmayinaeasethecellularem:ryofchoiesterolviaa specific receptor finally resulting in foam cell formatiOJl which has been seen for the fa time in imdiated human arteries bySheehan(43).
Is able to inhibit selectively PDGF-release from the pillelets. Subsequently. its proliferative 8I1d mitotic activity. which is known to be enhanced (14. 15. 44. 46). ated. Therefore. the capacity of the vessel wall to respond onto PDGF-liberatiOJl with an enhanced POIl- synthesis (8, SO) will be one of the key mechanisms in pathogenesis of the progressiOJl of radiatiOJl-induced vasculopatby (53). From basics it is, therefore. easy to understand. that the presence of elevated blood lipids 8I1d in particular of cholesterol would favour the foam cell formatiOJl (43) especially insofar as deaeased local POll-levels are favouring inIracellular cholesterol-synthesis and decrease the intracellular degndatiOJl (23).
Appl. Pbysiol. 2I: 833-838, 1984. W. C. J. Appl. Pbysiol. U: 1264-1269, 1986. M. I. Biull. Eksp. BioI. Med. 2i: 559-562, 1981. , Atzman, R. and Fuks, Z. Prostaglandins ~~: 263-279, 1983. L. and Kwock, L. Radiat. Res. i06: 171-181,1986. , Cahill, M. and Polgar, P. Prostaglandins ~2: 783-791, 1983. , Gardner, D. F. J. Appl. Physiol. 21t: 592-597, 1985. , Cromwell, M. and Firbas, W. Radiat. Res. 2I: 533-536, 1984. 36 61. 62. 63. 64. 65. 66. 67. 68. 69. 70. 71. 72. 73. 74. 75. 76. 77. 78. 79. SO. 81.